1.Objective To explore occlusion reconstruction by computer aided design (CAD) with functionally-generated pathway (FGP).
目的探索借助功能性?记录(functionally-generated pathway,FGP)技术进行口腔固定修复体计算机辅助设计(Computer aided Design,CAD)生理性建?的方法。
2.DATA SOURCES:A computer-based online search of Highwire was undertaken for the literature about the influence of nuclear factor κB in the proliferative disease published in English between January 1995 and December 2004 with the of "nuclear factor kappa B,vascular smooth muscle,proliferation and signal pathway".
资料来源:应用计算机检索highwire 1995-01/2004-12有关核因子κB对血管增殖性疾病影响的文献,检索词“nuclearfactor-Kappa B,vascular smooth muscle cell,proliferation,signal pathway”,并限定文章语言种类为English。
3.Some of them belonged to some important signal pathways related with cell proliferation and metastasis, such as DDK1, ILK, MMP-1 and COL11A1 in Wnt/β-catenin signal pathway, ILK in ILK signal pathway, EpHB2 in Eph-Ephrins signal pathway, ROCK2 in RhoA signal pathway, RANBP1 in RanGTPase signal pathway, and RBBP1 in RB/RBBP1 signal pathway.
其中包括一些非常重要的信号转导通路上的分子改变,如Wnt/β-catenin信号通路上的DKK1、ILK、MMP1、COL11A1,ILK信号通路上的ILK,Eph-Ephrins信号通路上的EpHB2,RhoA信号通路上的ROCK2,RanGTPase信号通路上的RANBP1,以及RB/RBBP1信号通路上的RBBP1等。
4.62 patients with atrioventricular nodal reentrant tachycardia (AVNRT) were treated with slow atrioventricular (AV) nodal pathway ablation in order to evaluate the effect of endpoint type A (no slow pathway conduction and no induclable AVNRT), B(residual slow pathway conduction but no induciable AVNRT and AVN echo) and C(residual slow pathway conduction and 1 to 3 AVN echo but no induciable AVNRT) on AV conduction and their relation with AVNRT recurrence.
62例慢-快型房室结折返性心动过速（AVNRT）患者接受慢径消融治疗，评价A型终点（阻断慢径且不再诱发AVNRT）、B型终点（保留慢径的传但不再诱发AVNRT和心房回波）和C型终点（保留慢径且能诱发1～3个心房回波）对房室传导功能的影响及与AVNT复发的关系。
5.The recent studies showed that SSR’s afferent nervous pathway was in the spinal cord and its central afferent pathway was closely correlated to the central afferent pathway of bladder sensation, which has provided us a new idea on the study of bladder sensory function.
最近研究表明:SSR 的传入神经通路在脊髓,其中枢传入通路与膀胱感觉的中枢传入通路密切关联,与躯体感觉通路不同。 这为我们研究膀胱感觉功能提供了新的思路。
6.Conclusion CaN signaling pathway plays an important role in FBs proliferation induced by IP 3, but it is not the single signaling pathway in regulating FBs proliferation, other signaling pathway may be also involved in FBs proliferation induced by IP 3.
CsA和Ver抑制IP3介导的FBs的CaN活性增高 ,Ver抑制IP3介导的FBs的PKC活性增高。 结论 CaN在IP3剌激的FBs增殖中起重要作用 ,但CaN信号通路不是IP3剌激FBs增殖的唯一信号通路 ,其它信号通路也可能参与了IP3剌激的FBs增殖。
7.As an important signal transducing pathway, the Janus kinase/signal transducer and activator of transcription (JAK/STAT) pathway is activated by various cytokines and growth factors. The JAK/STAT pathway plays an important role in growth, proliferation, differentiation, inflammation and apoptosis of cells.
Janu激酶/信号转导子和转录激活子(JAK/STAT)通路是近年来备受关注的一条细胞内重要的信号转导途径,参与介导细胞生长、增殖分化、炎症、凋亡等多种生理过程。
8.Methods The plasma levels of products of complement activation C4d (classic pathway), Bb (alternative pathway), 3bc (final common pathway) and C5b-9 (terminal complement complex) were measured in forty-four patients with CHF and sixteen normal volunteers with the age and sex matched.
方法 检测 4 4例心衰患者 (心衰组 )和 16例性别、年龄匹配的健康人 (对照组 )血浆补体经典激活途径产物C4d、替代激活途径产物Bb、终末共同途径产物C3bc和补体激活终末复合物C5b 9浓度。
9.Firstly, we investigate the expression of BH3 only protein Bim and Bad - the downstream effeector of the PDK/PKB pathway, during the canceriza-toin of endometrium. Then discuss the action of PKB during the formation of endometrial carcinoma, and its relationship with Bad, to expatiate the mechanism that the occurrence of endometrial carcinoma under the action of PI3K/PKB pathway. Finally, analyse the regulation of PTEN on the PDK/PKB pathway.
首先就PI3K／PKB途径下游效应因子——BH3 only protein(仅含BH3结构域蛋白)Bim、Bad在子宫内膜癌变过程中的表达进行研究，进而探讨PKB在子宫内膜癌发生过程中的作用及其与Bad的关系，研究PI3K／PKB途径激活促使子宫内膜癌发生的机制，最后分析PTEN对PI3K／PKB途径的调节作用。
10.After 24 hours, the flux of EMP pathway decreased by 33% averagely, the flux of HMP pathway increased by 76% averagely, and the flux of inosine synthesis pathway increased by 77% on average. The final inosine yield was improved from 17.44 g·L~-1 to 24 g·L~-1,by an increase of 37.6%.
在24h以后,EMP途径的代谢通量平均降低33%,HMP途径通量平均增加76%,肌苷合成途径通量平均增加77%,最终产苷由17.44g·L-1提高到24g·L-1,增幅达37.6%.
