1.The investigation confirmed that in rats necrosis tissue of acute necrotizing pancreatitis were all cultivated Escherichia coli and other negative Gram staining Bacilli, it is thus evident that controlling the gut origin infection can reduce the death of the SAP.
研究证实,在急性坏死型胰腺炎(acute necrotizing pancreatitis,ANP)大鼠坏死组织中均培养到大肠杆菌等革兰阴性杆菌,由此可见控制SAP时肠源性细菌感染(gut origin infection)理应能降低SAP的死亡率。 肠源性细菌感染的病理生理学基础,是肠黏膜屏障功能障碍。
2.SU_L(2)×U_1(1)×…×U_n(1) Electroweak Gauge Group and the Elimination of Monopole Conflict Between GUT and Big Bang Cosmology
SU_L(2)×U_1(1)×…×U_n(1)弱电规范群和GUT中磁单极矛盾的消除
3.4.Addition of 0.02%～0.5% chitosan in broiler chicken diets raised caecal aceticacid、butyric acid and total acid content, decreased caecal pH, and improved inner environment of intestine,addition of 0.02%～0.3% chitosan in diets accelerated growth of gut villi,which made gut villi become higher,thinner and density of gut villi become larger.
4.在肉仔鸡日粮中添加 0.02%～0.5%壳聚糖,可提高盲肠内容物乙酸、丁酸和总酸含量,降低肠道 pH 值,改善肠道内环境; 添加 0.02%～0.3%的壳聚糖可促进小肠绒毛生长发育,使绒毛变高、变细,密度加大,而添加 0.5%的壳聚糖,小肠绒毛结构没有得到改善。
4.and the abdominal mid gut(AMG)is mildly alkaline from pH 7.1 to 8.4 with the maximum pH observed at the junction with the hind gut.
腹部中肠（AMG）呈弱碱性，pH7．1－8．4，并且从前往后逐渐增高。
5.SCFA can enhance colon epithelial cell proliferation, may prevent gut atrophy associated with parenteral and low residue enteral feedings, and may also support the gut barrier function against the deleterious sequence of bacterial adhesion, colonization and translocation.
短链脂肪酸可促进结肠上皮细胞的增殖，防止肠外营养和低渣肠内营养引起的肠道粘膜萎缩，支持肠粘膜屏障功能，防止肠道细菌粘附、过度增殖和易位引起的损害。
6.5 APN activity of gut is higher than other tissues in H. armigera and Mythimna separata, and only the sample of gut can hybridize with Bt Cry1AcE toxin.
5 棉铃虫和粘虫肠部组织APN活性高于其他组织，并且只有肠部组织才与Bt CrylAcE毒素有结合。
7.At least there are three mechanisms of the early enteral feeding or nutrition: ①Reducing gut injury, improving blood flow of gastrointestinal tract, decreasing ischemia and reperfusion injury; ② Lessening the gut translocation of endotoxin, modulating activity of Kupffers cell; ③Lowering the levels of mediators and hormones.
早期肠道喂(营)养的机制至少有三：①减轻肠道损伤，改善胃肠道血流，减轻缺血再灌注损伤；②减少肠道内毒素移位，调理枯否细胞活化；③降低体内介质、激素水平。
8.Conclusions As important factors NO and ET 1 are pare of gut injury in ANP, and inhibition of the production of ET 1 and NO may decrease the gut injury in ANP dogs.
结论NO和ET?1是导致ANP时肠道损伤的重要因素，抑制ET?1的产生和减少早期NO的超量释放，可减轻ANP对肠道的损伤作用。
9.Ranging from 100 to 200 microns across, the width of several human hairs, these microscopic fossils are surprisingly complex and constitute almost a textbook example of a bilaterian, including the three major tissue layers (the endoderm, mesoderm and ectoderm familiar from high school biology texts), the presence of a gut with a mouth and anus, and paired coeloms (body cavities) surrounding the gut.
这些微化石的大小为100~200微米，相当于几根头发的宽度，??强雌鹄闯銎娓丛樱??壹蛑本拖窳讲喽猿贫?锏慕炭剖榘悖?故玖烁髦痔蒯纾喊?ㄈ?鲋饕?淖橹?悖?谂卟恪⒅信卟慵巴馀卟悖?际侵醒??锟伪局惺煜さ拿?剩??昂罅?涌诓坑敫孛诺某η唬?η煌饣刮?浦?啥缘奶迩弧
10.Transplanting gut microbes from these fat mice into lean, germ-free mice led to greater gains in body fat than transplanting gut microbes from normal mice.
接受取自肥胖老鼠体内微生物的无菌老鼠比接受普通老鼠的体重增加更多。
